Understanding your health
Simplified Information
Verified Sources

Lumbar Osteoarthritis

Lumbar osteoarthritis—or osteoarthritis of the lower back—is one of the most common findings on medical images. Yet it remains one of the most poorly understood conditions. Seeing "arthritis" or "degenerative changes" on an X-ray or MRI report can be frightening. It suggests damage that cannot be repaired. It...
4.9
Verified by Google

Lumbar Osteoarthritis

Written by:
Scientifically reviewed by:

You have just received an X-ray or MRI report. The words "arthritis," "degenerative changes," or "wear and tear" may have caused you concern. They suggest permanent damage. They suggest that the pain will only get worse. It is normal to be concerned when you read these terms on a medical report.

Here's the good news: osteoarthritis is a normal part of aging, just like gray hair or wrinkles.¹ Studies show that 60% of adults aged 40 have visible osteoarthritis on imaging, but many have no pain.⁹ Structural changes do not necessarily correspond to symptoms.²

What research shows:
  • People with severe osteoarthritis on their scans may have no pain at all.³
  • Those with minimal changes may experience significant discomfort.³
  • Function can improve significantly with physical therapy, despite visible changes on imaging.⁷⁴

This guide explains what lumbar osteoarthritis really is, why images do not determine your future, and how physical therapy can help you maintain function and reduce pain.

What is lumbar spine osteoarthritis?

Lumbar osteoarthritis is the normal wear and tear of cartilage in the facet joints and disc spaces in your lower back. It causes pain, stiffness, and reduced mobility. This normal degenerative process affects most adults over the age of 60. However, it can be managed very well with the right treatment and lifestyle changes.

Osteoarthritis develops when cartilage wears away over time. Cartilage is the smooth tissue that covers the surfaces of joints. In the lumbar spine, it mainly affects the facet joints.⁴ These are small pairs of joints located at the back of each vertebra. They control how your spine moves.

Faceted joints contain fluid and cartilage. This makes them similar to the joints in your knees or hips.⁵ When the cartilage wears away, the bones can develop small bumps called osteophytes (bone spurs). The joint space also becomes narrower. These changes show up on medical images. They are often called "degenerative disc disease" or "spondylosis" when they also affect the discs between your vertebrae.⁶

Osteoarthritis vs. inflammatory arthritis

Lumbar osteoarthritis is a condition caused by wear and tear. It is not an inflammatory arthritis like rheumatoid arthritis.⁷ Degenerative arthritis develops due to repeated stress and aging. Inflammatory arthritis occurs when your immune system attacks your joints.⁸ This difference is important because the treatment approaches differ greatly. Our education about your condition helps you demystify osteoarthritis and adopt the right behaviors.

Studies show that lumbar osteoarthritis is extremely common. Imaging reveals osteoarthritis of the facet joints in more than 60% of adults aged 40 to 49. Almost all adults over the age of 80 have it.⁹ Yet many of these people report no back pain. This clearly shows that structural changes are not the same as symptoms.¹⁰ To understand your lower back pain, you need to look beyond the images.

What causes osteoarthritis to develop in the lower back?

Lumbar osteoarthritis develops due to repeated mechanical stress, aging, genetics, past injuries, and lifestyle factors. All of these elements affect the cartilage in the joints. The cartilage breaks down. The bones remodel. Inflammation occurs. This creates structural changes that vary greatly from person to person. These changes have very little to do with the level of pain you feel.

Daily mechanical stress

Your lower back supports heavy loads during your daily activities. This is especially true at the L4-L5 and L5-S1 levels, where most movement occurs.¹¹ Repeated loading over decades slowly wears down the cartilage surfaces. This triggers bone changes. Jobs that involve heavy lifting, long periods of sitting, or vibrations can accelerate this process. However, the relationship is complex and not completely predictable.¹²

The role of genetics

Genetics play a big role in the risk of osteoarthritis. Studies on twins suggest that about 50 to 65% of the risk of osteoarthritis is inherited. It affects the composition of your cartilage. It influences your bone density. It changes how you respond to inflammation.¹³ If your parents developed osteoarthritis, you are more likely to develop it too. But it does not determine whether you will experience pain.

The impact of past injuries

Past injuries can trigger or accelerate arthritic changes. Vertebral fractures, herniated discs, or ligament sprains change how joints move and bear weight. This can trigger earlier cartilage degradation in the affected areas.¹⁴ However, many people with extensive injury histories never develop painful osteoarthritis. Others with no history of injury develop it anyway.

Factors you can control

You can control certain risk factors: obesity, lack of exercise, and smoking.

  • Body weight: Extra weight increases pressure on the facet joints. This accelerates cartilage wear.¹⁵
  • Movement: Surprisingly, complete rest also promotes osteoarthritis. It reduces cartilage nutrition. Cartilage needs movement to stay healthy.¹⁶
  • Smoking: Smoking slows down tissue healing. It can accelerate degeneration due to its effects on blood vessels.¹⁷

Disc degeneration and facet joint osteoarthritis usually develop together. As discs lose height and water with age, facet joints undergo altered loading.¹⁸ This link means that "disc disease" and "facet osteoarthritis" often occur together.

What are the symptoms of lumbar osteoarthritis?

Symptoms include morning stiffness lasting less than 30 minutes, dull back pain that worsens with activity, reduced flexibility, and sometimes a grinding sensation during movement. If you recognize these symptoms, rest assured: they are very common and respond well to active treatment. The pain usually remains in the lower back without major symptoms in the legs, unless stenosis develops.

Typical morning stiffness

Morning stiffness is typical of osteoarthritis. After long periods of rest, your joints feel tight. Moving seems difficult. But symptoms usually improve within 15 to 30 minutes once you start moving.¹⁹ This is different from inflammatory arthritis, where stiffness lasts for an hour or more. The stiffness comes from temporary thickening of the joint fluid and reduced lubrication of the joints during rest.²⁰

Osteoarthritis vs. inflammatory arthritis: how to tell them apart

FeatureOsteoarthritis (degenerative)Inflammatory arthritis
Morning stiffnessLess than 30 minutesMore than an hour
CauseWear and tear and agingImmune system
EvolutionSlow, gradualVariable, in episodes
ImprovementWith a light movementWith the initial rest

Pain patterns during the day

Pain patterns follow mechanical patterns. Discomfort increases with prolonged standing, walking, or leaning backward. These movements put stress on the facet joints.²¹ Sitting may provide relief at first (it takes the load off the facet joints), but sitting for too long often causes discomfort. The pain is usually described as deep, dull, or stiff, rather than sharp or burning.²² This pattern of pain aggravated by extension is explained in detail in our guide to directional preferences.

Pure osteoarthritis vs. nerve compression

Unlike nerve compression problems, osteoarthritis alone rarely causes major symptoms in the legs. The pain usually remains in the lower back. It may spread to the buttocks or upper thighs, but it does not follow specific nerve pathways.²³ However, when bone spurs or thickened ligaments press on the nerve passages in the spine, spinal stenosis can develop.

Functional limitations

Functional limitations develop slowly. You may notice that you are less able to bend forward or backward. You may have difficulty getting up from a chair. These limitations are often more related to pain avoidance and muscle protection than to actual joint damage.²⁴ This means that they can improve significantly with the right treatment, even when structural changes remain.

Key point The severity of symptoms does not correspond to the severity of osteoarthritis on images. Research consistently shows a weak link between X-ray findings and pain levels.²⁵

10 mini-tips to understand your pain

Those who have had the greatest impact on my patients' lives. 1 per day, 2 min.

How is lumbar osteoarthritis diagnosed?

The diagnosis combines clinical examination, which shows reduced mobility and local sensitivity, with X-ray results. However, imaging results do not always correspond to symptoms. This makes clinical assessment essential for treatment planning. Structural results alone cannot be relied upon.

Your physical therapist or doctor will look for several typical signs during the examination. They will check your spinal mobility and note any reduction in range of motion when you bend forward, backward, or rotate.²⁷ They will palpate to find sensitivity above the facet joints. These are located about 2 to 3 cm from the middle of your lower back.²⁸ Backward bending movements usually cause pain, while forward bending can provide relief.²⁹ Our approach to movement rehabilitation specifically targets these restrictions.

What changes on X-rays indicate lumbar osteoarthritis?

X-rays show the structural signs of osteoarthritis:

  • Narrowing of the joint space as the cartilage becomes thinner
  • Formation of osteophytes (bone spurs) at the edges of joints
  • Subchondral sclerosis (increased bone density under the cartilage)
  • Subchondral cysts (fluid-filled pockets in the bone)

Radiologists often classify the severity of osteoarthritis from mild to severe based on these findings.³⁰ CT scans provide more detailed views of bone changes. MRI shows cartilage, joint inflammation, and soft tissue problems.³¹

Why don't imaging results always match symptoms?

The discrepancy between imaging and symptoms is one of the most important concepts in managing lumbar osteoarthritis. Large population studies show that people without symptoms often have the same imaging findings as those with severe pain.³² One major study found osteoarthritis of the facet joints in 89% of adults over the age of 60. However, many reported no back pain.³³

This discrepancy occurs because pain depends on several factors beyond structure:

  • Muscle conditioning
  • Movement patterns
  • Sensitivity of the nervous system
  • Psychological factors
  • Sleep quality
  • Stress levels³⁴
This understanding should be deeply reassuring. The osteoarthritis visible on your scans does not condemn you to worse pain or disability. Treatment focuses on factors you can change, not on reversing structural changes (which is impossible anyway).

How does physical therapy treat osteoarthritis, and does exercise help?

Physical therapy treatments:

Physical therapy combines manual therapy, targeted exercises, and education to manage symptoms. Treatment focuses on keeping you functional rather than reversing osteoarthritis.

Joint mobilization and manipulation improve facet joint mobility.³⁶ Soft tissue work reduces muscle tension.³⁷

Therapeutic exercise forms the basis of management. Strength and endurance exercises strengthen the core and hip muscles that support your spine.³⁸ Flexibility exercises maintain your range of motion.³⁹ Aerobic exercise promotes cartilage health.⁴⁰

Education about the science of pain empowers you to take control. Our personalized education program demystifies chronic pain.⁴¹ The multimodal approach consistently shows better results than a single treatment.⁴³

Exercise as the most effective treatment:

Exercise is the most effective treatment for osteoarthritis. It reduces pain and improves function despite joint changes. Cartilage obtains its nutrients during joint compression and decompression. Regular exercise keeps cartilage healthier than rest. Appropriate loading protects cartilage.

Several types of exercise are beneficial for osteoarthritis: stabilizing muscle exercises for the core, flexibility exercises, and aerobic activities.The McKenzie approach offers a structured method of directional exercises.

The dose matters: keep discomfort below 3-4 out of 10 during exercise.⁵¹ Progress is slow.⁵³ A little discomfort is normal.⁵⁴ The benefits last as long as you continue the exercise.⁵⁷

Make an appointment for a comprehensive evaluation and personalized treatment plan.

What lifestyle changes help manage lumbar osteoarthritis?

Key changes include maintaining a healthy weight to reduce joint stress, staying physically active with low-impact activities, improving your posture and workspace layout, using heat for stiffness, and pacing your activities to avoid flare-ups.

Weight management

Weight management profoundly affects osteoarthritis symptoms. Every additional kilogram of body weight increases the pressure on the lumbar joints, several times that weight during movement.⁵⁸ Weight loss of just 5 to 10% of body weight can greatly reduce pain and improve function.⁵⁹

Choice of activities

Choosing and modifying activities keeps you moving without aggravating symptoms. Low-impact activities such as swimming, water aerobics, cycling, or using an elliptical trainer provide cardiovascular benefits while reducing stress on your joints.⁶⁰ A comprehensive posture analysis reveals imbalances that aggravate your osteoarthritis.

Workspace layout

Workplace design reduces ongoing stress during daily activities. A well-adjusted desk and chair reduce long periods in one position. Alternate between sitting and standing throughout the day.⁶² Ourworkplace ergonomics assessment service identifies adjustments specific to your work situation.

Use of heat

Heat works well to reduce stiffness and improve movement. Morning showers, heating pads, or warm pools help prepare tissues for activity.⁶⁴ Heat is usually most helpful before activity.

Activity measurement

Pacing and conserving energy prevent symptom flare-ups. Alternate between harder and easier activities. Take regular movement breaks. Increase activity levels slowly.⁶⁶ Pacing does not mean avoiding activity: it means distributing activity intelligently.

Need professional advice?

Our physical therapists can assess your condition and offer you a personalized treatment plan.

Book an appointment

How does lumbar osteoarthritis progress over time?

Osteoarthritis typically progresses slowly over years, with periods of stability and occasional flare-ups. Although structural changes continue, symptoms do not always worsen at the same rate. Many people maintain good function through active management. Symptoms often stabilize rather than constantly worsen.

Studies on natural history show that the progression of lumbar osteoarthritis varies greatly and is often unpredictable.⁶⁷ Some people show rapid progression on X-rays with minimal change in symptoms. Others have stable imaging results but changing symptoms.⁶⁸ This shows that structural progression and symptom progression are partly separate processes.

Managing flare-ups

Flare-ups (periods of increased pain and stiffness) sometimes occur in most people with osteoarthritis. This is normal and does not mean that your condition is getting worse. They are often triggered by a new activity, poor sleep, or stress.⁶⁹ Strategies include temporary changes in activities (not complete rest), increased use of heat or ice, and continuing gentle movement.⁷¹

Factors affecting progression

Ongoing obesity, smoking, complete lack of activity, and poor muscle conditioning can accelerate symptoms.⁷² On the other hand, maintaining a healthy weight, staying physically active, and managing other conditions such as diabetes or depression can slow the progression of symptoms.⁷³

The outlook for maintaining function is generally good with active management. Long-term studies show that most people with lumbar osteoarthritis maintain their functional abilities over 5 to 10 years, especially when they engage in regular exercise and physical therapy.⁷⁴

When should other treatments be considered, and how can facet syndrome be distinguished?

Other treatment options:

Consider other treatments when physical therapy alone does not adequately control symptoms after 8–12 weeks. Facet injections offer temporary relief.⁷⁶ Radiofrequency ablation provides longer-lasting relief for some patients.⁷⁷ NSAIDs are effective in the short term but carry gastrointestinal risks.⁸⁰ Surgery remains a last resort.⁸¹

If your osteoarthritis is the result of a workplace accident, the CNESST covers 100% of the treatment costs. Our telerehabilitation service offers comprehensive remote sessions.

Distinction from facet syndrome:

Osteoarthritis involves chronic degenerative changes that are visible on imaging. Facet syndrome describes acute irritation, often without structural changes.⁸⁶ Osteoarthritis develops slowly over months or years. Facet syndrome often occurs suddenly. Many patients have both conditions simultaneously.

How can you regain control of your lumbar osteoarthritis?

Our physical therapists create personalized, evidence-based programs to reduce pain, improve mobility, and keep you active. The osteoarthritis visible on your images will remain, but symptoms can improve significantly through physical therapy, exercise, and lifestyle changes.

We help you move from fear of diagnosis to confident self-management. If your condition is the result of a traffic accident, our services are covered by the SAAQ.

Make an appointment to plan your assessment and create a personalized program.

---

References

1. Brinjikji W, Luetmer PH, Comstock B, et al. Systematic literature review of imaging features of spinal degeneration in asymptomatic populations. AJNR Am J Neuroradiol. 2015;36(4):811-816.

2. Kalichman L, Li L, Kim DH, et al. Facet joint osteoarthritis and low back pain in the community-based population. Spine. 2008;33(23):2560-2565.

3. Suri P, Miyakoshi A, Hunter DJ, et al. Does lumbar spinal degeneration begin with the anterior structures? BMC Musculoskeletal Disorders. 2011;12:202.

4. Bogduk N. Clinical and Radiological Anatomy of the Lumbar Spine. 5th ed. Edinburgh: Churchill Livingstone; 2012.

5. Jaumard NV, Welch WC, Winkelstein BA. Spinal facet joint biomechanics and mechanotransduction. J Biomech Eng. 2011;133(7):071010.

6. Kirkaldy-Willis WH, Wedge JH, Yong-Hing K, Reilly J. Pathology and pathogenesis of lumbar spondylosis and stenosis. Spine. 1978;3(4):319-328.

7. Goldring MB, Goldring SR. Osteoarthritis. J Cell Physiol. 2007;213(3):626-634.

8. Felson DT. Osteoarthritis as a disease of mechanics. Osteoarthritis Cartilage. 2013;21(1):10-15.

9. Suri P, Miyakoshi A, Hunter DJ, et al. Does lumbar spinal degeneration begin with the anterior structures? BMC Musculoskeletal Disorders. 2011;12:202.

10. Kalichman L, Li L, Kim DH, et al. Facet joint osteoarthritis and low back pain in the community-based population. Spine. 2008;33(23):2560-2565.

11. Nachemson AL. Disc pressure measurements. Spine. 1981;6(1):93-97.

12. Bakker EW, Verhagen AP, Lucas C, et al. Daily spinal mechanical loading as a risk factor for acute non-specific low back pain. Eur Spine J. 2007;16(1):107-113.

13. Battié MC, Videman T, Levälahti E, et al. Genetic and environmental effects on disc degeneration. Spine. 2008;33(25):2801-2808.

14. Adams MA, Roughley PJ. What is intervertebral disc degeneration, and what causes it? Spine. 2006;31(18):2151-2161.

15. Shiri R, Karppinen J, Leino-Arjas P, et al. The association between obesity and low back pain: a meta-analysis. Am J Epidemiol. 2010;171(2):135-154.

16. Holm S, Maroudas A, Urban JP, et al. Nutrition of the intervertebral disc. Connect Tissue Res. 1981;8(2):101-119.

17. Behrend C, Prasarn M, Coyne E, et al. Smoking cessation related to improved patient-reported pain scores. J Bone Joint Surg Am. 2012;94(23):2161-2166.

18. Fujiwara A, Lim TH, An HS, et al. The effect of disc degeneration and facet joint osteoarthritis on segmental flexibility. Spine. 2000;25(23):3036-3044.

19. Hootman JM, Helmick CG. Projections of US prevalence of arthritis and associated activity limitations. Arthritis Rheum. 2006;54(1):226-229.

20. Buckwalter JA, Saltzman C, Brown T. The impact of osteoarthritis: implications for research. Clin Orthop Relat Res. 2004;(427 Suppl):S6-15.

21. Manchikanti L, Boswell MV, Singh V, et al. Prevalence of facet joint pain in chronic spinal pain. BMC Musculoskeletal Disorders. 2004;5:15.

22. Schwarzer AC, Aprill CN, Derby R, et al. Clinical features of patients with pain stemming from the lumbar zygapophysial joints. Spine. 1994;19(10):1132-1137.

23. Bogduk N. International Spinal Injection Society guidelines for spinal injection procedures. Clin J Pain. 1997;13(4):285-302.

24. Gellhorn AC, Katz JN, Suri P. Osteoarthritis of the spine: the facet joints. Nat Rev Rheumatol. 2013;9(4):216-224.

25. Kalichman L, Li L, Kim DH, et al. Facet joint osteoarthritis and low back pain in the community-based population. Spine. 2008;33(23):2560-2565.

26. O'Sullivan P. It's time for change with the management of non-specific chronic low back pain. British Journal of Sports Medicine. 2012;46(4):224-227.

27. Cook C, Hegedus E, Showalter C, Sizer PS Jr. Coupling behavior of the lumbar spine. J Manipulative Physiol Ther. 2006;29(7):554-558.

28. Dreyfuss P, Michaelsen M, Pauza K, et al. The value of medical history and physical examination in diagnosing sacroiliac joint pain. Spine. 1996;21(22):2594-2602.

29. Laslett M, Öberg B, Aprill CN, McDonald B. Zygapophysial joint blocks in chronic low back pain. BMC Musculoskeletal Disorders. 2004;5:43.

30. Kellgren JH, Lawrence JS. Radiological assessment of osteoarthrosis. Ann Rheum Dis. 1957;16(4):494-502.

31. Weishaupt D, Zanetti M, Hodler J, Boos N. MR imaging of the lumbar spine. Radiology. 1998;209(3):661-666.

32. Brinjikji W, Luetmer PH, Comstock B, et al. Systematic literature review of imaging features of spinal degeneration. AJNR Am J Neuroradiol. 2015;36(4):811-816.

33. Kalichman L, Li L, Kim DH, et al. Facet joint osteoarthritis and low back pain. Spine. 2008;33(23):2560-2565.

34. Moseley GL, Butler DS. Fifteen years of explaining pain. J Pain. 2015;16(9):807-813.

35. Fairbank JC, Pynsent PB. The Oswestry Disability Index. Spine. 2000;25(22):2940-2952.

36. Bronfort G, Haas M, Evans R, et al. Effectiveness of manual therapies. Chiropr Osteopat. 2010;18:3.

37. Bialosky JE, Bishop MD, Price DD, et al. The mechanisms of manual therapy. Man Ther. 2009;14(5):531-538.

38. Searle A, Spink M, Ho A, Chuter V. Exercise interventions for the treatment of chronic low back pain. Clin Rehabil. 2015;29(12):1155-1167.

39. Hayden JA, van Tulder MW, Tomlinson G. Systematic review: strategies for using exercise therapy. Ann Intern Med. 2005;142(9):776-785.

40. Dunn KM, Jordan KP, Croft PR. Contributions of prognostic factors for poor outcome. Eur J Pain. 2011;15(3):313-319.

41. Louw A, Diener I, Butler DS, Puentedura EJ. The effect of neuroscience education on pain. Archives of Physical Medicine and Rehabilitation. 2011;92(12):2041-2056.

42. Vlaeyen JW, Linton SJ. Fear-avoidance and its consequences. Pain. 2000;85(3):317-332.

43. van Middelkoop M, Rubinstein SM, Verhagen AP, et al. Exercise therapy for chronic nonspecific low-back pain. Best Pract Res Clin Rheumatol. 2010;24(2):193-204.

44. Foster NE, Anema JR, Cherkin D, et al. Prevention and treatment of low back pain. Lancet. 2018;391(10137):2368-2383.

45. Urban JP. The role of the physicochemical environment in determining disc cell behavior. Biochem Soc Trans. 2002;30(Pt 6):858-864.

46. Belavý DL, Albracht K, Bruggemann GP, et al. Can exercise positively influence the intervertebral disc? Sports Med. 2016;46(4):473-485.

47. Holm S, Maroudas A, Urban JP, et al. Nutrition of the intervertebral disc. Connect Tissue Res. 1981;8(2):101-119.

48. Hides JA, Richardson CA, Jull GA. Multifidus muscle recovery. Spine. 1996;21(23):2763-2769.

49. Marshall PW, Kennedy S, Brooks C, Lonsdale C. Pilates exercise or stationary cycling for chronic nonspecific low back pain. Spine. 2013;38(15):E952-959.

50. Hurwitz EL, Morgenstern H, Chiao C. Effects of recreational physical activity. American Journal of Public Health. 2005;95(10):1817-1824.

51. Smith BE, Hendrick P, Smith TO, et al. Should exercises be painful in the management of chronic musculoskeletal pain? Br J Sports Med. 2017;51(23):1679-1687.

52. American College of Sports Medicine. ACSM's Guidelines for Exercise Testing and Prescription. 10th ed. Philadelphia: Wolters Kluwer; 2018.

53. Nijs J, Roussel N, van Wilgen CP, et al. Thinking beyond muscles and joints. Man Ther. 2013;18(2):96-102.

54. Smith BE, Hendrick P, Smith TO, et al. Should exercises be painful? British Journal of Sports Medicine. 2017;51(23):1679-1687.

55. Henchoz Y, Kai-Lik So A. Exercise and nonspecific low back pain. Joint Bone Spine. 2008;75(5):533-539.

56. Jordan JL, Holden MA, Mason EE, Foster NE. Interventions to improve adherence to exercise. Cochrane Database Syst Rev. 2010;(1):CD005956.

57. Bassett SF. The assessment of patient adherence to physical therapy rehabilitation. N Z J Physical Therapy. 2003;31(2):60-66.

58. Shiri R, Karppinen J, Leino-Arjas P, et al. The association between obesity and low back pain. Am J Epidemiol. 2010;171(2):135-154.

59. Christensen R, Bartels EM, Astrup A, Bliddal H. Effect of weight reduction in obese patients. Ann Rheum Dis. 2007;66(4):433-439.

60. Dundar U, Solak O, Yigit I, et al. Clinical effectiveness of aquatic exercise. Spine. 2009;34(14):1436-1440.

61. Hurwitz EL, Morgenstern H, Chiao C. Effects of recreational physical activity. American Journal of Public Health. 2005;95(10):1817-1824.

62. Robertson M, Amick BC III, DeRango K, et al. The effects of an office ergonomics training. Appl Ergon. 2009;40(1):124-135.

63. Claus AP, Hides JA, Moseley GL, Hodges PW. Different ways to balance the spine. Spine. 2009;34(6):E208-214.

64. French SD, Cameron M, Walker BF, et al. Superficial heat or cold for low back pain. Cochrane Database Syst Rev. 2006;(1):CD004750.

65. Nadler SF, Weingand K, Kruse RJ. The physiological basis of cryotherapy and thermotherapy. Pain Physician. 2004;7(3):395-399.

66. Murphy SL, Smith DM, Clauw DJ, Alexander NB. The impact of momentary pain and fatigue. Arthritis Rheum. 2008;59(6):849-856.

67. Suri P, Hunter DJ, Rainville J, et al. Presence and extent of severe facet joint osteoarthritis. Osteoarthritis Cartilage. 2013;21(9):1199-1206.

68. Fujiwara A, Lim TH, An HS, et al. The effect of disc degeneration and facet joint osteoarthritis. Spine. 2000;25(23):3036-3044.

69. Hawker GA, Stewart L, French MR, et al. Understanding the pain experience. Osteoarthritis Cartilage. 2008;16(4):415-422.

70. Parry E, Ogollah R, Peat G. Significant pain variability in persons with knee osteoarthritis. BMC Musculoskeletal Disorders. 2017;18(1):80.

71. Bannuru RR, Osani MC, Vaysbrot EE, et al. OARSI guidelines for non-surgical management. Osteoarthritis Cartilage. 2019;27(11):1578-1589.

72. Sandmark H, Nisell R. Validity of five common manual neck pain provoking tests. Scandinavian Journal of Rehabilitation Medicine. 1995;27(3):131-136.

73. Foster NE, Anema JR, Cherkin D, et al. Prevention and treatment of low back pain. Lancet. 2018;391(10137):2368-2383.

74. Hartvigsen J, Hancock MJ, Kongsted A, et al. What low back pain is and why we need to pay attention. Lancet. 2018;391(10137):2356-2367.

75. Foster NE, Anema JR, Cherkin D, et al. Prevention and treatment of low back pain. Lancet. 2018;391(10137):2368-2383.

76. Manchikanti L, Pampati V, Fellows B, Bakhit CE. The diagnostic validity of facet joint nerve blocks. Current Review in Pain. 2000;4(5):337-344.

77. Maas ET, Ostelo RW, Niemisto L, et al. Radiofrequency denervation for chronic low back pain. Cochrane Database Syst Rev. 2015;(10):CD008572.

78. American College of Rheumatology. Recommendations for medical management of osteoarthritis. Arthritis Rheum. 2000;43(9):1905-1915.

79. Zhang W, Moskowitz RW, Nuki G, et al. OARSI recommendations for management of osteoarthritis. Osteoarthritis Cartilage. 2008;16(2):137-162.

80. Chou R, McDonagh MS, Nakamoto E, Griffin J. Analgesics for Osteoarthritis. Rockville: AHRQ; 2011.

81. Mirza SK, Deyo RA. Systematic review of randomized trials comparing lumbar fusion surgery. Spine. 2007;32(7):816-823.

82. Jacobs W, Van der Gaag NA, Tuschel A, et al. Total disc replacement for chronic back pain. Cochrane Database Syst Rev. 2012;(9):CD008326.

83. Deyo RA, Mirza SK, Martin BI, et al. Trends, complications, and charges for lumbar spinal stenosis surgery. JAMA. 2010;303(13):1259-1265.

84. Vickers AJ, Vertosick EA, Lewith G, et al. Acupuncture for chronic pain. J Pain. 2018;19(5):455-474.

85. Astin JA. Why patients use alternative medicine. JAMA. 1998;279(19):1548-1553.

86. Manchikanti L, Boswell MV, Singh V, et al. Prevalence of facet joint pain. BMC Musculoskeletal Disorders. 2004;5:15.

87. Schwarzer AC, Aprill CN, Derby R, et al. Clinical features of lumbar facet syndrome. Spine. 1994;19(10):1132-1137.

88. Schwarzer AC, Wang SC, Bogduk N, et al. Prevalence and clinical features of lumbar facet joint pain. Ann Rheum Dis. 1995;54(2):100-106.

89. Gellhorn AC, Katz JN, Suri P. Osteoarthritis of the spine: the facet joints. Nat Rev Rheumatol. 2013;9(4):216-224.

90. Friedrich KM, Nemec S, Peloschek P, et al. The prevalence of lumbar facet joint edema. Skeletal Radiology. 2007;36(8):755-760.

91. Kalichman L, Li L, Kim DH, et al. Facet joint osteoarthritis and low back pain. Spine. 2008;33(23):2560-2565.

92. Cohen SP, Raja SN. Pathogenesis, diagnosis, and treatment of lumbar facet joint pain. Anesthesiology. 2007;106(3):591-614.

93. Gellhorn AC, Katz JN, Suri P. Osteoarthritis of the spine: the facet joints. Nat Rev Rheumatol. 2013;9(4):216-224.

94. Bronfort G, Haas M, Evans R, et al. Effectiveness of manual therapies. Chiropr Osteopat. 2010;18:3.

Other conditions

Cervical osteoarthritis
Hip Osteoarthritis (Coxarthrosis)

Hip osteoarthritis is a normal wear and tear of the hip joint. Osteoarthritis is often described as the wearing away of cartilage between our bones. While this is true, it involves more than just cartilage. Cartilage is a tissue that acts like a cushion between the surfaces of our bones, allowing our joints to glide smoothly and move with ease.

Knee Osteoarthritis (Gonarthrosis)

It is a normal wear and tear of the knee joint. Osteoarthritis is often described as the wearing away of cartilage between our bones. While this is true, it involves more than just cartilage. Cartilage is a tissue that acts like a cushion between the surfaces of our bones, allowing our joints to glide smoothly and move with ease.

Hip Bursitis

A bursa is like a small, very thin, fluid-filled sac found in several joints throughout the body. This small sac acts as a cushion in the joint and lubricates structures that are exposed to more friction.

Shoulder Bursitis

It is an inflammation of the subacromial bursa in the shoulder joint.

Bursite à l'épaule : Traitement et guérison en physio
Shoulder capsulitis (frozen shoulder)

It is a tissue that surrounds the shoulder and allows the shoulder bone to stay in place within the joint. The capsule helps to stabilize the joint.

Cervicalgia

Cervicalgia is a general term to describe neck pain that does not have a specific cause, such as an accident or sudden movement. Cervicalgia is therefore synonymous with ''I have a pain in my neck and nothing in particular happened''.

Cervicobrachialgia or cervical radiculopathy

In both injuries, there is pain felt in the neck that then radiates into the arm, or vice versa.

Adductor Strain

It is a significant stretch or tear of the muscle fibers in the groin or inner thigh muscles.

Hamstring Strain

It is a significant stretch or tear of the muscle fibers in the hamstring muscles located at the back of the thigh.

Calf Strain

This is a significant stretch or tear of the muscle fibers in the calf muscles (soleus and gastrocnemius).

Book an appointment now

We offer a triple quality guarantee: optimized time, double physiotherapy assessment, and ongoing expertise for effective care tailored to your needs.

A woman receives a rejuvenating neck massage in a peaceful and serene professional spa setting.
Main contents
Background image:
A woman receives a rejuvenating neck massage in a peaceful and serene professional spa setting.

Our clients' satisfaction is our priority.

At Physioactif, excellence defines our approach. But don't take our word for it, see what our patients are saying.

4.7/5
Fast Relief
4.9/5
Expertise
5/5
Listening

Discover our physiotherapy clinics

We have multiple locations to better serve you.

Book an appointment now

A man receives a relaxing muscle massage with a yellow strap support.
Main contents
Background image:
A man receives a relaxing muscle massage with a yellow strap support.