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Lumbo-cruralgia

Femoral nerve pain affects about 3-4% of people who experience pain radiating into the leg.¹ It originates from the lumbar spine. It is much less common than sciatica (sciatic nerve pain), but just as bothersome. If you are experiencing this pain in the front of your thigh, rest assured: it is a condition that responds very well to ph...
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Illustration of lumbar vertebrae and an irritated femoral nerve, from the "Lumbosacral Pain" guide by Physioactif

Lumbo-cruralgia

Written by:
Chloé Roy
Scientifically reviewed by:
Stéphanie Desjardins

Your lower back pain is now radiating down the front of your thigh. You feel a burning or tingling sensation in your groin or down the front of your leg. Climbing stairs is becoming difficult, and getting up from a chair takes effort. This isn't the same as sciatica, and you're wondering what's going on.

Good news: Cruralgia responds very well to physical therapy.¹ Cruralgia is significantly less common than sciatica among conditions that cause pain radiating down the leg, although its exact prevalence has not been clearly established in the literature. What science tells us about femoral nerve pain (cruralgia):
  • The femoral (crural) nerve runs along the front of the thigh; this is different from the sciatic nerve, which runs along the back
  • Cruralgia improves with proper treatment, but the recovery time varies greatly depending on the severity and cause of the compression of the femoral nerve.
  • Neural mobilization specific to the femoral nerve is a key treatment technique.

This guide explains why your thigh hurts and how to recover. To understand the broader context, consult our complete guide to back pain.

What is femoral neuralgia and which nerve does it affect?

Cruralgia is pain that runs down the femoral nerve. It starts in the lumbar spine, passes through the groin, and travels down the front of the thigh. This pain affects the L2, L3, and L4 nerve roots, unlike sciatica, which affects the back of the leg.

The nerve originates within the psoas muscle. It emerges beneath the inguinal ligament (in the groin). The femoral nerve provides sensation and strength to the front of the thigh and the inner side of the leg.³ It is one of the largest nerve branches in the lower back. It controls the quadriceps (the muscles at the front of the thigh). This muscle group allows you to extend your knee.⁴

The key difference between cruralgia and sciatica lies in the nerve’s path. Cruralgia affects the upper lumbar nerve roots (L2–L4). It causes pain on the front and inner side of the thigh. Sciatica affects the lower lumbar nerve roots (L4–S1). This causes pain in the back of the leg that extends below the knee.² Understanding the structure of your spine helps explain why back pain can cause such different symptoms. It depends on which nerve is affected.

Comparative table showing key differences:

To learn more about these two conditions, consult our complete guide to sciatica. You will see how they differ in presentation and treatment.

Cruralgia is less common than sciatica. This rarity is linked to herniated discs in the upper lumbar spine. These herniations are less common than those in the lower lumbar spine (at the L4-L5 and L5-S1 levels).⁶ However, cruralgia can be just as painful and debilitating as sciatica. It primarily affects activities that require bending the hip and extending the knee. For example, climbing stairs becomes difficult. Getting up from a chair requires more effort. Getting out of bed can be painful.⁷ To understand the full range of lumbar radiculopathies, our guide explores the different levels of nerve compression. You’ll see how each level affects your function.

What causes femoral neuralgia to develop?

Cruralgia results from compression or irritation of the upper lumbar nerve roots (L2–L4). The most common cause is a herniated disc at the L2–L3 or L3–L4 levels. Spinal stenosis, osteoarthritis of the joints, and, more rarely, tumors can also cause it.

In a herniated disc, the soft center of the disc (the nucleus pulposus) protrudes through the outer fibrous layers (the annulus fibrosus), much like the filling of a doughnut oozing out through a crack. The protruding material compresses the adjacent nerve root. This occurs in the spinal canal or the lateral recess (the narrow passages for the nerves).⁸

A piece of good news that is often overlooked: a herniated disc—the most common cause of cruralgia—can resolve on its own over time, without treatment. A herniated disc has a real ability to heal.

Upper lumbar disc herniations are less common than lower lumbar disc herniations. The L2-L3 and L3-L4 segments experience slightly less mechanical stress compared to the L4-L5 and L5-S1 levels when you bend over or lift objects.⁶

However, when high lumbar herniations do occur, they often cause severe symptoms. The spinal canal is relatively narrow at these levels. The nerve roots are located in an anterior position. This makes them vulnerable to compression by disc herniations.⁸ To understand the mechanisms of disc compression, discover our complete guide to lumbar disc herniation. You will see how discs degenerate and compress nerves.

Stenosis of the lateral recess at the upper lumbar levels is the second most common cause. It occurs mainly in older adults.⁹ The lateral recess is the narrow bony canal through which the nerve roots exit the spinal canal. They then become peripheral nerves.⁹ Stenosis (narrowing) develops through several processes. Loss of disc height is one of them. Hypertrophy (enlargement) of the facet joints is another. Thickening of the ligamentum flavum and the formation of bone spurs also contribute.⁹

These age-related changes gradually reduce the space available for the nerve roots. This creates compression that produces femoral neuralgia symptoms. When you are standing or walking, symptoms often worsen. When you are sitting or leaning forward, you often feel relief.⁹ The progressive narrowing of the spinal canal is a major cause of nerve pain in people over 50. Learn how to identify and treat this condition in our complete guide to spinal stenosis.

Facet joint problems at the L2-L3, L3-L4, or L4-L5 levels can contribute to cruralgia. Synovial cysts (small fluid-filled sacs) can directly compress the nerve root. Inflammation and swelling can also cause indirect compression.¹⁰ Osteoarthritis of the facet joints and synovial cysts occur more frequently in the mobile segments of the lumbar spine. These cysts can protrude into the lateral recess or the intervertebral foramen (the openings for the nerves). They compress the adjacent nerve roots.¹⁰ Facet-related cruralgia often presents with two types of pain. You experience pain that radiates down the leg. You also experience localized pain at the affected segment.¹⁰ Joint degeneration of the spine is a major contributing factor. Check out our guide on lumbar osteoarthritis to understand how this condition progresses. You’ll see how it affects the nervous structures.

List of less common causes:

Less common causes of femoral neuralgia include:

  • Spinal tumors (primary or metastatic) affecting the upper lumbar vertebrae or nerve roots
  • Epidural abscess or discitis (infections of the disc space)
  • Spondylolisthesis in the upper lumbar spine (when a vertebra slips)
  • Diabetic amyotrophy, which primarily affects the femoral nerve
List of risk factors:

Risk factors that increase your chances of developing femoral neuralgia:

  • Age over 50
  • Occupations that require repeated trunk rotations or lifting heavy loads
  • Previous lumbar surgery that causes degeneration of the adjacent segment
  • Smoking, which affects disc nutrition
  • Diabetes, which increases the risk of neuropathy (nerve disease)¹²

What are the characteristic symptoms of femoral neuralgia?

If you recognize these symptoms, know that this is a common condition that responds well to treatment. The main symptom of cruralgia is pain on one side only. It originates in the lumbar spine and travels through the groin area. The pain travels down the front or front-inner side of the thigh.⁵ People describe the pain as sharp, burning, electric, or aching. It follows the distribution of the L2, L3, or L4 dermatomes—the areas of skin controlled by each nerve root. The specific nerve root that is compressed determines the exact pattern of pain.¹³ This anterior distribution contrasts sharply with the pain in the back of the leg seen in sciatica. The location of the pain is very helpful for diagnosis.⁵ Pain from an irritated nerve root in the lower back does not always follow the classic expected path of the nerve. Often, it is not limited to the precise territory of the affected nerve. An atypical pain pattern is therefore not, in and of itself, a sign of severity. Radiating nerve pain requires a thorough evaluation to identify the affected nerve root. Explore the characteristics of lumbosciatica. You’ll be able to compare posterior versus anterior pain patterns.

Table showing dermatomal patterns:

Groin pain is a characteristic symptom of cruralgia. This helps distinguish it from sciatica.¹⁴ You may feel discomfort in the groin. You may experience a pulling sensation. The pain may seem to originate from inside the hip joint. This often leads to an incorrect initial diagnosis. People tend to suspect a hip problem rather than nerve compression in the spine.¹⁴

Motor weakness affecting the quadriceps is a concerning sign of femoral neuralgia, impacting your daily activities.¹⁵ The femoral nerve controls the quadriceps muscles, which are the four muscles at the front of the thigh responsible for extending the knee.⁴ When the L2-L4 nerve roots experience significant compression, you develop weakness that affects knee extension, hip flexion, and bringing the thigh closer to the body.¹⁵

In practical terms, this means that climbing stairs becomes difficult. You have trouble getting up from low chairs or the toilet. Walking uphill requires more effort. Your knee may become unstable when it has to support your weight while walking.⁷ Severe weakness in the quadriceps can cause your knee to give way suddenly—a phenomenon known as knee buckling. This creates a risk of falling and significantly limits your ability to function.¹⁵

Sensory symptoms include numbness, tingling, or altered sensation. This affects the front of the thigh and can extend to the inner leg and foot. The specific nerve root determines the extent of these sensory changes.¹³ These sensory changes follow patterns that correspond to the level of the compressed nerve root. Experienced clinicians can often predict the site of compression based on the sensory distribution.¹³ Reflex changes can also occur. The patellar reflex (knee-jerk reflex) might be diminished or absent, as this reflex tests the function of the L3-L4 nerve root.¹⁶

Pain that feels like a pinched nerve sometimes originates in the muscles. Muscle tension points can mimic true nerve pain, and this aspect is often treatable. It’s worth having it evaluated.

List of aggravating and relieving factors:

Positions and movements that often worsen femoral neuralgia pain:

  • Prolonged standing (which puts stress on the spine)
  • Hip extension (which stretches the femoral nerve)
  • Walking for extended periods
  • Lying flat on your back with legs extended
  • Activities that require repeated hip extension

Positions and movements that often relieve femoral neuralgia pain:

  • Sitting with hips flexed
  • Lie on your unaffected side with the affected hip slightly bent.
  • Forward flexion positions that open the lateral recess
  • Slight bend at the hips and knees.

Night pain is common. When you lie flat, your hip is in relative extension. This can increase tension on the femoral nerve. It can also maintain compression, depending on the underlying cause.¹⁸

10 Quick Tips for Understanding Your Pain

The ones that have made the biggest difference in my patients' lives. 1 a day, 2 minutes.

How do healthcare professionals diagnose femoral nerve pain?

The diagnosis is based primarily on clinical evaluation. MRI imaging can confirm the extent of compression, but it is very rarely necessary in cases of cruralgia.

The clinical diagnosis of cruralgia begins with a complete medical history—the story of your pain. We characterize the location, nature, and path of the pain. We identify what makes it worse and what provides relief. We determine when it started and how long it has lasted. We assess any associated symptoms.¹⁹ We evaluate your gait. We look for weakness in the quadriceps. This may be noticeable when you carefully descend stairs. Your gait may be altered. You may avoid certain movements to prevent pain.¹⁹ An assessment of your posture may reveal positions you adopt to minimize nerve tension. For example, a slight flexion of the trunk, or keeping the affected hip flexed.¹⁹

The neurological examination systematically assesses muscle strength, sensory function, and reflexes. It identifies the specific nerve root level involved.¹⁶ The motor test assesses hip flexion strength (L1-L2), thigh adduction (L2-L3), knee extension (L3-L4), and foot elevation (L4-L5).¹⁶ The sensory examination maps the distribution of altered sensation, numbness, or abnormal sensations. It determines whether the L2, L3, or L4 dermatomes are affected.¹³ The patellar reflex test assesses the function of the L3-L4 nerve root. Diminished or absent reflexes indicate significant nerve compression at these levels.¹⁶

What clinical tests identify femoral nerve involvement?

The femoral nerve stretch test is the primary test for cruralgia.²⁰ It is also known as the inverted straight-leg raise. This test is performed with you lying face down. Your hip is passively extended and your knee is flexed, which stretches the femoral nerve and places stress on the L2-L4 nerve roots.²⁰ The test is positive when it reproduces your typical pain in the front of the thigh. Studies report a sensitivity of approximately 84–95% for upper lumbar disc herniation.²⁰ This test is different from the straight leg raise test used for sciatica. The latter stresses the sciatic nerve and the lower lumbar nerve roots.²¹

The sagging test can be modified to assess tension on the femoral nerve. You sit with your spine flexed and your hip extended. Adding knee flexion increases the stress on the femoral nerve.²² If symptoms improve when you extend your neck during this maneuver, it suggests neurogenic pain rather than a musculoskeletal cause.²² Palpation is also performed on the segments of the lumbar spine. This identifies tender areas. Muscle guarding or spasm in the back muscles often accompanies acute nerve irritation.¹⁹

How does femoral nerve pain differ from hip problems?

Distinguishing cruralgia from a hip problem is a significant diagnostic challenge. Both conditions can cause pain in the groin and the front of the thigh.²³ Hip osteoarthritis, labral tears, greater trochanter pain syndrome, and femoral head anterior displacement (or hip instability, or impingement syndrome) all affect the hip or the surrounding structures. These conditions can refer pain to the front of the thigh.²³ In contrast, a hip problem generally causes pain localized to the groin, the side of the hip, or the front of the thigh. The pain does not extend below the knee. There are no sensory changes in specific areas of the skin or changes in reflexes. Specific hip examination tests are positive.²³

The hip range-of-motion test helps distinguish between these conditions. A hip problem typically presents with restricted and painful hip internal rotation. Flexion may be limited. Combined movements (the FABER and FADIR tests) are painful. Cruralgia preserves relatively normal hip joint mobility, unless muscle protection limits movement.²⁴ People with a hip problem often report mechanical symptoms. For example, clicking, catching, or a sensation of locking. They have difficulty putting on shoes and socks. They experience pain specifically localized to the hip during standing activities.²⁴ People with cruralgia more often describe neurogenic symptoms—such as burning or electric-shock-like sensations and numbness. The patterns of radiation follow dermatomes rather than joint distribution.⁵

Imaging plays a confirmatory role in diagnosis when clinical findings suggest the need to visualize nerve compression.²⁵ Magnetic resonance imaging (MRI) provides excellent soft-tissue detail. It shows herniated discs, spinal stenosis, and nerve root compression. It also shows soft-tissue masses that may cause cruralgia.²⁵ However, as with sciatica, imaging is not necessary for initial management. Exceptions include cases with red flags, such as progressive weakness, bowel or bladder dysfunction, or suspected tumor or infection. Imaging is also performed if symptoms persist beyond 6–8 weeks despite appropriate treatment.²⁶

If you have an MRI, don’t be discouraged by the results. About 20–35% of people without symptoms show lumbar disc abnormalities on MRI.²⁷ This underscores the fact that imaging results must align with your clinical presentation for an accurate diagnosis. CT scans provide excellent bone detail. They assess spinal stenosis or narrowing of the lateral recess. However, they provide less soft-tissue detail than an MRI.²⁸

Nerve roots have plenty of space where they exit the spine: the opening through which they pass is much wider than the nerve root itself. This is one of the reasons why compression visible on imaging does not always cause pain.

How does physiotherapy effectively treat femoral nerve pain?

Physical therapy treatment includes neural mobilization for the femoral nerve—that is, specialized exercises that help the nerve glide more smoothly. There are specific exercises to reduce compression of the nerve root. Manual therapy improves spinal mobility. Progressive strengthening of the affected muscles is essential.

Evidence-based physical therapy for cruralgia employs several treatment approaches. These are specifically tailored to the pathology of the upper lumbar nerve root and the course of the femoral nerve.²⁹ The treatment strategy progresses through overlapping phases rather than following a rigid timeline. In the more acute phase, the focus is on managing pain and nerve irritation. Stabilization exercises and postural correction begin right from the start, not at a fixed point in time. Strengthening then progresses according to each individual’s tolerance, with the goal of restoring function and preventing recurrence.²⁹

For complete information on how physiotherapy treats back pain, our guide explains the methods physiotherapists use. You'll learn how we treat spinal nerve root compression and how to safely return to normal activities.

Femoral nerve mobilization techniques are specialized procedures. They promote the gliding of the nerve through the surrounding tissues. They reduce mechanical sensitivity and support natural healing processes.³⁰ You will be guided through progressive nerve mobilization exercises. The path of the femoral nerve is alternately lengthened and shortened. The exercises are performed within a comfortable range of motion. They do not worsen your symptoms.³⁰ Nerve mobilization is a specialized therapeutic approach. It requires clinical expertise. Learn about our approach to nerve treatment. You’ll explore neural gliding techniques and their clinical applications.

Typical progressions include femoral nerve glides performed lying on your side, extending the hip with knee flexion. This advances to femoral nerve stretch exercises performed lying on your stomach. Eventually, you'll progress to mobilizations in standing or functional positions as your tolerance improves.³⁰ Research shows that neural mobilization combined with other interventions significantly reduces nerve pain and improves function.³¹

Physical therapy techniques that gently move and slide the nerve—known as neural mobilization—help reduce pain and disability when pain radiates down the leg from the lower back.

Positioning and movement strategies help identify your directional preference—that is, specific positions or movements that reduce or centralize your symptoms.³² For cruralgia, many people experience relief with forward-flexion positions. However, some respond better to neutral or extension-based approaches. This depends on their specific condition.³² A systematic evaluation identifies your particular directional preference. Treatment focuses on repeated movements or sustained positions in the preferred direction. This promotes the centralization of symptoms, where pain shifts from the leg toward the spine. Symptom centralization is associated with better outcomes.³² Unlike sciatica, where extension predominates, cruralgia exhibits more variable directional preferences. This requires an individualized assessment.³³ Identifying directional preferences is based on proven systematic assessment principles. Learn how the McKenzie approach uses repetitive movements to identify and treat nerve compression.

Manual therapy techniques tailored to the upper lumbar segments include spinal mobilization at the L2-L3 and L3-L4 levels. Manipulation is used when appropriate and not contraindicated by the patient’s condition. Manual therapy addresses joint restrictions, reduces muscle spasm, and improves spinal mobility. It also modulates pain through neurophysiological mechanisms.³⁴ Scientific evidence supports manual therapy combined with exercise rather than either intervention alone, particularly for subacute and chronic conditions.³⁵ Manual techniques specific to the upper lumbar levels require advanced training. Visit our page on joint mobilizations and manipulations. You’ll see how these precise techniques restore spinal mobility.

Gradual strengthening of the quadriceps can be an important component of cruralgia rehabilitation, particularly in severe cases of compression, but it is not always necessary. Nerve compression often causes muscle weakness and atrophy (loss of muscle mass). This persists even after the pain resolves.³⁶ Strength training begins with isometric quadriceps contractions—contractions without movement. Straight-leg raises are performed. This places a load on the muscles without significant movement of the hip or knee. Training progresses to partial-range exercises under load. For example, wall squats and stair climbs with controlled depth. This progresses to full-range functional exercises. For example, squats, lunges, and single-leg activities.³⁶ Regaining muscle strength requires a systematic and individualized progression. Explore ourstrength training program. You’ll see how we adapt the progressive load to your level of recovery.

Concurrent core stabilization training provides spinal support during activities. It reduces mechanical stress on affected segments and prevents compensatory movement patterns that could slow down recovery.³⁷ Core stability protects vulnerable spinal structures. Discover our approach to stabilizing muscle exercises. These exercises target the deep muscles essential for lumbar support.

Pain management strategies include education on the mechanisms of pain. Advice is provided on adjusting activity levels. Patients are taught positions that can relieve symptoms. Thermal therapies (heat or ice, depending on the phase) can be helpful. Patient education is important: the relationship between body positions and symptoms is explained, and patients are reassured about the safety of the activity. This significantly improves outcomes and reduces fear-based avoidance behaviors that slow recovery.³⁹

What is the recovery timeline for cruralgia?

Cruralgia generally responds well to appropriate physical therapy, but the recovery time varies greatly from person to person. Mild cases tend to recover more quickly than those with severe compression, but no studies allow for the establishment of a precise timeframe. Severe compression accompanied by weakness may require a long recovery period, and a full return of strength is not always guaranteed. Starting treatment early significantly improves the speed and completeness of recovery.

Rest assured: the natural course of cruralgia is generally favorable. Most cases show significant improvement within 6–12 weeks of symptom onset, when appropriate treatment is initiated.⁴⁰ Studies on upper lumbar disc herniations report that approximately 60–75% of people experience substantial pain relief. They regain functional recovery with treatment that includes physical therapy.⁴⁰ Acute cruralgia (symptoms lasting less than 6 weeks) generally responds more quickly than chronic cases, often showing noticeable improvement within 2–4 weeks of starting treatment.⁴¹

Recovery progresses through predictable phases. This corresponds to the processes of nerve healing.⁴² Initial improvement involves a reduction in pain and the centralization of symptoms. This occurs as inflammation subsides. Mechanical compression decreases through positioning, therapeutic exercises, and natural healing.⁴² Sensory recovery generally precedes motor recovery. Numbness and tingling gradually resolve. This happens as nerve root compression decreases.⁴² Motor strength recovery follows a slower timeline, particularly when significant weakness has developed. Nerve reinnervation of muscle fibers takes time, as the axon (the long part of the nerve) regrows and the neuromuscular junction is restored.⁴² Severe weakness of the quadriceps may take 8–16 weeks or longer to achieve substantial recovery of strength. This timeline applies even after the pain has improved significantly.⁴³

When monitored for a year, people with a herniated disc compressing a nerve root see their symptoms subside over time. The various symptoms improve at roughly the same rate.

List of positive prognostic indicators:

Factors indicating faster recovery:

  • Younger age
  • Shorter duration of symptoms before starting treatment
  • Absence of severe motor weakness
  • Presence of a clear directional preference with symptom centralization
  • Good adherence to prescribed exercises and activity modifications⁴⁴
List of factors associated with prolonged recovery:

Factors that can slow down recovery:

  • Older age with significant spinal degeneration
  • Severe baseline weakness (particularly grade 2/5 or less)
  • A prolonged duration of symptoms before the start of treatment is associated with a poorer prognosis.
  • Psychosocial factors including depression or catastrophic thinking (believing it's worse than it is)
  • Presence of litigation or workers' compensation involvement⁴⁵

Starting physical therapy early speeds up recovery. It can prevent acute pain from progressing to chronic pain.⁴⁶ Beginning treatment without undue delay after symptoms appear is good clinical practice. Early treatment likely prevents secondary complications. For example, muscle deconditioning, altered movement patterns, central sensitization (overly sensitive nerves), and fear-based avoidance behaviors.⁴⁶ Even long-standing cruralgia can improve with comprehensive physical therapy, although recovery becomes more uncertain as symptoms persist. However, recovery times tend to be longer. More intensive rehabilitation may be required to address secondary factors that have developed during the chronic phase.⁴⁷

Considerations regarding motor recovery are particularly important in cruralgia. Weakness in the quadriceps significantly affects your daily activities and quality of life.⁴³ You should expect gradual improvements in strength over 2–4 months, once the nerve compression has been adequately treated. However, full recovery to pre-injury strength levels may require 4–6 months of progressive resistance training.⁴³ Significant muscle weakness that does not improve despite treatment warrants an immediate surgical consultation, rather than waiting. Prolonged denervation (a nerve disconnected from the muscle for an extended period) can result in permanent muscle changes. This reduces the potential for recovery.⁴⁸

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What positions provide relief from cruralgia pain?

Postural adjustments are an essential self-management strategy for cruralgia. Specific postures reduce nerve root compression. They decrease tension on the femoral nerve. They minimize pain.⁴⁹ Understanding the biomechanical principles underlying symptom relief allows you to make informed decisions about positioning throughout the day and night.⁴⁹

Sleeping positions have a significant impact on cruralgia symptoms. Lying on your back with your legs extended places the hip in relative extension. This can increase tension on the femoral nerve. It can also maintain compression, depending on your specific condition.¹⁸ Lying on your unaffected side often provides substantial relief. Keep the affected hip in a slight flexion. Place a pillow between your knees. This reduces nerve tension.⁵⁰ Some people find relief by lying on their affected side with the leg slightly bent. This can reduce compression of the lateral recess.⁵⁰

Lying on your stomach generally worsens the symptoms of cruralgia. This position puts the hips in extension, which increases the lumbar curve. Both of these factors can increase pressure on the nerve roots.⁵⁰ Raising the head of the bed slightly while lying on your back may provide relief for some people. Placing a pillow under your knees while lying on your back can also help. This reduces pressure on the nerves.⁵⁰

Adjustments to sitting posture play a crucial role in managing cruralgia symptoms, particularly for people whose work or lifestyle involves prolonged sitting.⁵¹ Although sitting generally places the hip in flexion (which reduces tension on the femoral nerve), the position of the lumbar spine significantly affects nerve root compression.⁵¹ Maintaining adequate lumbar support with a cushion or rolled-up towel often helps. This promotes a slight lumbar curve without excessive extension.⁵¹ The chair should be high enough to allow the feet to rest flat on the floor. The hips and knees should be flexed at approximately 90 degrees.⁵¹ Taking standing breaks every 20–30 minutes prevents prolonged sitting. This promotes circulation. However, these breaks should involve gentle movement, not prolonged standing, which can worsen symptoms.⁵¹

Strategies for standing and walking differ from those for managing sciatica. Cruralgia symptoms often worsen with prolonged standing or walking. This occurs due to the load on the spine and hip extension during walking.⁵² Limiting continuous standing or walking to tolerable levels is helpful. Using assistive devices when necessary reduces the load. Incorporating rest periods while sitting or in hip-flexed positions can also help manage symptoms during necessary standing activities.⁵² Some people benefit from leaning slightly forward while standing. This reduces the lumbar curve and opens the lateral recess. However, this position should not be maintained for too long, as it can create other biomechanical problems.⁵²

List of activities to temporarily avoid:

Activities to temporarily avoid during acute femoral neuralgia:

  • Running (which creates repeated hip extension and spinal loading)
  • Aggressive stretching of the quadriceps or hip flexors that puts too much tension on the femoral nerve
  • Prolonged standing on hard surfaces
  • Activities that require repeated hip extension, such as climbing stairs or step-ups
  • Heavy lifting that increases pressure within the disc⁵³

As your symptoms improve, these activities are gradually reintroduced. You will be provided with guidance, appropriate modifications, and progressive loading.⁵³

Individual variation in symptom response to positioning highlights the importance of self-experimentation. Professional evaluation is also important to identify your specific directional preference and optimal positions.³² What provides relief for one person with femoral neuralgia may not help another. This depends on the underlying pathology, location of compression, and individual biomechanics. Personalized positional advice from your physiotherapist is essential for effective symptom management.³²

How does femoral neuralgia management differ from sciatica?

Femoral neuralgia affects the front of the thigh, requiring different nerve mobilization techniques than sciatica, which affects the back. Exercise positions favor flexion with a hip flexion bias. Functional rehabilitation focuses on quadriceps strength rather than calf muscles.

Understanding the distinctions between femoral neuralgia and sciatica management ensures appropriate treatment. Treatment must target the correct anatomical structures and nerve pathways.⁵ Although both conditions involve lumbar nerve pain, the specific techniques and exercise selections differ based on nerve distribution and affected muscle groups.⁵

Differences in treatment techniques center on nerve mobilization approaches. These target the specific nerve involved.³⁰ Mobilization of the femoral nerve for cruralgia uses hip extension positions combined with knee flexion. This places stress on the anterior nerve pathway. It is performed in the side-lying, prone, or standing positions.³⁰ This contrasts with sciatic nerve mobilization for sciatica. Sciatic nerve mobilization uses hip flexion with knee extension, in the supine or seated positions. This places stress on the posterior nerve pathway.³⁰

Exercises that are beneficial for sciatica could potentially worsen cruralgia. The reverse is also true. This underscores the critical importance of an accurate diagnosis before beginning nerve-specific treatments.³⁰

Variations in exercise selection reflect the different nerve root levels, affected muscle groups, and positional preferences between the two conditions.³³ Femoral neuralgia rehabilitation emphasizes quadriceps strengthening. Exercises include straight leg raises, wall squats, and step-downs, addressing L2-L4 related weakness. Sciatica rehabilitation prioritizes strengthening muscles that flex and extend the ankle, addressing L5-S1 related deficits.⁵⁴

Stretching the hip flexors should be done with caution in cases of cruralgia. Aggressive stretching of the iliopsoas can increase tension on the femoral nerve, which may worsen symptoms. Preference-based exercises reveal different patterns. People with sciatica primarily demonstrate an extension bias. They benefit from prone push-ups and extension exercises. People with cruralgia show more variable responses. Many prefer neutral or flexion-based positions.³³

Rehabilitation for cruralgia prioritizes activities that require quadriceps function. For example, climbing stairs and standing up from a chair, which require knee control during the stance phase of walking. Rehabilitation for sciatica emphasizes calf function for propulsion during walking. The focus is on ankle control for foot roll-off. Toe flexion strength is addressed.⁷

Functional progression for cruralgia includes gradual stair training, progressive weight-bearing knee extension exercises, and balance activities that challenge quadriceps control. Functional training for sciatica, on the other hand, emphasizes calf raises, walking on the heels, and activities that require plantar flexion strength in the ankle.⁵⁴

Cruralgia and sciatica generally respond well to physical therapy, although no direct, quantified comparison of success rates between these conditions is available in the literature. However, cruralgia may show a slightly slower motor recovery when significant quadriceps weakness develops, due to the functional importance of this muscle group for walking.⁴⁰ Both conditions follow a favorable natural course. Most cases improve substantially within 6–12 weeks. However, individual recovery timelines vary. This depends on the severity of the condition, patient-specific factors, and adherence to treatment.⁴⁰

When is advanced intervention necessary for femoral neuralgia?

Red flags requiring urgent medical evaluation in femoral neuralgia are rare (less than 1% of low back pain cases). However, you must seek immediate emergency care if you have:⁵⁵

  • Progressive motor weakness that worsens over days to weeks despite treatment
  • Sudden onset of severe quadriceps weakness that prevents you from climbing stairs or rising from chairs
  • Loss of bladder or bowel control (suggests cauda equina syndrome)
  • Saddle anesthesia (numbness in the genital area)
  • Symptoms in both legs

These symptoms suggest severe spinal cord or cauda equina compression. This requires an urgent evaluation and potential surgical decompression.⁵⁵

Rest assured: cauda equina syndrome is rare. It most commonly presents with bilateral symptoms of sciatica caused by a central disc herniation. However, severe upper lumbar pathology can occasionally affect several nerve roots simultaneously.⁵⁶

The surgical indications for cruralgia follow guidelines similar to those for sciatica. Surgery is considered when treatment fails after an appropriate trial period (usually 6–12 weeks) and significant functional limitations persist.⁵⁷ Cruralgia most often results from a lumbar disc herniation. Approximately 60% of these herniations resolve on their own, which explains why many cases are resolved without surgery. Decisions regarding surgery involve shared decision-making between you and the surgeon, who weighs the severity of your symptoms, the functional impact on your quality of life, your preferences, and the surgical risks against the expected outcomes.⁵⁷ Progressive motor weakness is a stronger indication for surgery than pain alone. Prolonged nerve compression can lead to permanent muscle denervation. Full recovery may not be possible even after successful decompression.⁴⁸

The duration of conservative treatment before considering surgery generally ranges from 6 to 12 weeks. This applies to people without progressive weakness or red flags.⁵⁸ Research shows that most cases of cruralgia that will improve with conservative treatment show significant progress within this timeframe. People who do not respond after 12 weeks are less likely to experience substantial improvement without intervention.⁵⁸ However, even chronic cruralgia lasting more than 3–6 months can respond to comprehensive physical therapy, particularly when previous treatment was inadequate or not optimally targeted.⁴⁷

A cortisone injection in the lower back is slightly more effective than a placebo at reducing leg pain in the short term. However, the effect is small and may not always be significant for the patient. It is not a long-term treatment.

List of surgical options:

Surgical options for femoral neuralgia depend on the underlying condition causing nerve compression:⁵⁹

  • Microdiscectomy: removes the herniated portion of the disc that is compressing the nerve root at the L2-L3 or L3-L4 levels
  • Laminectomy or decompressive laminotomy: widens the lateral recess or intervertebral foramen when stenosis causes compression
  • Facet joint cyst excision: treats compression caused by synovial cysts that protrude into the spinal canal

Success rates for various procedures show that appropriate surgical candidates achieve short-term pain relief rates of 80–90%. In contrast, long-term results (2–5 years) show more modest differences.⁶¹ The SPORT trial on lumbar disc herniation found something interesting. Although patients who underwent surgery experienced a faster initial improvement, the outcomes converged between the surgical and conservative groups at 2–4 years.⁶² This research suggests that surgery accelerates recovery. However, it does not necessarily improve long-term outcomes compared to high-quality physical therapy for people who can tolerate the longer recovery timeline associated with conservative treatment.⁶²

For sciatica, the sister condition of cruralgia, surgery provides slightly faster relief at first, but this benefit is no longer significant after six months: those treated without surgery catch up with those who underwent surgery.

Postoperative rehabilitation with physical therapy is essential following surgical decompression for cruralgia. Surgery addresses mechanical compression. However, it does not correct biomechanical factors. It does not correct the movement patterns or muscle deficits that contributed to the problem. These factors also develop during the symptomatic period.⁶³ Comprehensive post-surgical physical therapy significantly improves functional outcomes and reduces the risk of recurrence by systematically addressing these factors.⁶³

How can you prevent femoral neuralgia from recurring?

Prevention focuses primarily on maintaining flexibility in the lumbar spine and strengthening the core stabilizing muscles. It is also important to maintain good posture while sitting and lifting. Regular exercise and ergonomic adjustments significantly reduce the risk of recurrence.

Core stabilization is the foundation of femoral neuralgia prevention. It provides dynamic spinal support, reducing forces on intervertebral discs and protecting nerve roots during daily activities.³⁷ Core musculature includes deep stabilizers like the multifidus and transversus abdominis, as well as superficial muscles such as the rectus abdominis, obliques, erector spinae, and quadratus lumborum. They work together to control spinal position and transfer loads efficiently.³⁷

Research shows that people who maintain regular core exercise programs after their nerve pain has resolved have significantly lower recurrence rates compared to those who stop exercising once their symptoms improve.⁶⁴ A balanced development of flexibility and strength is helpful, achieved through proper stretching (avoiding excessive tension on the femoral nerve) and gradual strengthening as recovery allows. This helps maintain optimal hip mechanics and reduces the risk of recurrence.⁶⁵

Optimizing posture and ergonomics during sitting, standing, and lifting activities minimizes cumulative spinal stress, which contributes to disc degeneration and recurrent nerve compression.⁶⁶ Good lifting mechanics include maintaining a neutral spinal alignment, squatting to lift objects from the floor, keeping loads close to your body, avoiding twisting while holding weights, and dividing large loads into smaller transports.⁶⁶

Ergonomic workplace setup for office workers includes chair height that allows feet to be flat on the floor, lumbar support that maintains natural curves, a monitor positioned at eye level, and regular position changes every 30 minutes.⁶⁶ For manual laborers, work task analysis and modification help reduce repetitive lumbar flexion, rotation, or heavy lifting, protecting the spine from cumulative trauma.⁶⁶

Weight management affects the risk of cruralgia recurrence. Excess body weight increases the mechanical load on the lumbar discs and nerve roots.⁶⁷ Each kilogram of excess weight generates several times that force throughout the spine during activities and while standing.⁶⁷ In addition, obesity is associated with increased systemic inflammation. It is linked to metabolic dysfunction that affects disc health and the body’s ability to heal.⁶⁷ Achieving and maintaining a healthy weight through a balanced diet and regular physical activity provides significant protective benefits.⁶⁷

Addressing the underlying biomechanical abnormalities identified during the physical therapy evaluation is perhaps the most important individualized prevention strategy.⁶⁸ Movement pattern dysfunctions may require retraining of motor control. Muscular imbalances may require specific strengthening programs. Joint restrictions may require ongoing maintenance of mobility.⁶⁸ Systematically correcting the contributing factors removes the mechanical stresses that caused your initial episode of cruralgia. This substantially reduces the likelihood of recurrence.⁶⁸

For comprehensive prevention programs and ongoing management advice, consult our physiotherapy treatment guide. It provides detailed strategies for maintaining spinal health and preventing recurrent nerve compression after recovering from femoral neuralgia.

List of lifestyle modifications:

Lifestyle modifications that support long-term prevention:

  • Quitting smoking significantly improves disc nutrition and healing capacity (smoking affects blood flow to the intervertebral discs and slows down nerve healing)⁶⁹
  • Regular physical activity, including aerobic exercise, strength training, and flexibility work, maintains overall spinal health and reduces the risk of injury⁷⁰
  • Stress management through techniques like mindfulness, adequate sleep, and relaxation practices reduces muscle tension and pain amplification, which can contribute to pain recurrence or becoming chronic⁷¹

Ready to address your femoral nerve pain?

Our physiotherapists at Physioactif specialize in evaluating and treating femoral nerve pain. We develop personalized programs to relieve femoral nerve compression and restore your normal function.

Don't let femoral nerve pain limit your ability to climb stairs, get up from chairs, or participate in activities you enjoy. Radiating femoral nerve pain requires expert evaluation and specialized treatment. Discover how our team effectively treats back pain and radiating leg pain. We use evidence-based approaches.

Contact Physioactif today to schedule your comprehensive evaluation. Our experienced team will confirm your diagnosis, identify the specific cause of your femoral nerve compression, and develop an individualized treatment plan. Through specialized nerve mobilization techniques, targeted exercises, manual therapy, and progressive strengthening, we will guide you toward a complete recovery. Take the first step towards relief by booking an appointment now.

References

  1. Tarulli AW, Raynor EM. Lumbosacral radiculopathy. Neurol Clin. 2007;25(2):387-405.
  2. Dionne N, Adefolarin A, Kunzelman D, et al. Differential diagnosis and management of crural neuralgia versus sciatica: a narrative review. Cureus. 2021;13(8):e17340.
  3. Romanowski CA, Beletsky V, Perry J. Anatomy, Abdomen and Pelvis, Femoral Nerve. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2024 [cited 2025 Jan 10]. Available: https://www.ncbi.nlm.nih.gov/books/NBK557507/
  4. Bowden JL, Heales LJ, Tabor A, et al. Exploring the relationship between femoral nerve neurodynamics and pain provocation and self-reported disability in patients with acute lateral hip pain. Musculoskelet Sci Pract. 2019;44:102050.
  5. Ropper AH, Zafonte RD. Sciatica. N Engl J Med. 2015;372(13):1240-1248.
  6. Beutler WJ, Fredrickson BE, Murtland A, Sweeney CA, Grant WD, Baker D. The natural history of spondylolysis and spondylolisthesis: 45-year follow-up evaluation. Spine. 2003;28(10):1027-1035.
  7. Hislop HJ, Avers D, Brown M. Daniels and Worthingham's Muscle Testing: Techniques of Manual Examination and Performance Testing. 9th ed. St. Louis, MO: Elsevier Saunders; 2013.
  8. Matsui H, Kanamori M, Ishihara H, Yudoh K, Naruse Y, Tsuji H. Familial predisposition for lumbar degenerative disc disease: a case-control study. Spine. 1998;23(9):1029-1034.
  9. Lurie J, Tomkins-Lane C. Management of lumbar spinal stenosis. BMJ. 2016;352:h6234.
  10. Siebenrock KA, Schoeniger R, Ganz R. Anterior femoro-acetabular impingement due to acetabular retroversion: treatment with periacetabular osteotomy. J Bone Joint Surg Am. 2003;85(2):278-286.
  11. Tracy JA, Dyck PJB. The spectrum of diabetic neuropathies. Phys Med Rehabil Clin N Am. 2008;19(1):1-26.
  12. Shiri R, Karppinen J, Leino-Arjas P, Solovieva S, Viikari-Juntura E. The association between smoking and low back pain: a meta-analysis. Am J Med. 2010;123(1):87.e7-35.
  13. Waxenbaum JA, Futterman B. Anatomy, Back, Lumbar Vertebrae. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2024 [cited 2025 Jan 10]. Available: https://www.ncbi.nlm.nih.gov/books/NBK459278/
  14. Santaguida PL, McGill SM. The psoas major muscle: a three-dimensional geometric study. J Biomech. 1995;28(3):339-345.
  15. Powers CM, Ward SR, Chan LD, Chen YJ, Terk MR. The effect of bracing on patellofemoral joint stress during free and fast walking. Am J Sports Med. 2004;32(1):224-231.
  16. Bono CM, Ghiselli G, Gilbert TJ, et al. An evidence-based clinical guideline for the diagnosis and treatment of cervical radiculopathy from degenerative disorders. Spine J. 2011;11(1):64-72.
  17. Nachemson A. The load on lumbar disks in different positions of the body. Clin Orthop Relat Res. 1966;45:107-122.
  18. Goldstein B, Young J, Escobedo E. Rotator cuff repairs: Are there any differences between acute and chronic tears? Arthroscopy. 2001;17(9):961-966.
  19. Cook CE, Wilhelm M, Cook AE, Petrosino C, Quillen W. Clinical tests for screening and diagnosis of hip, knee, and ankle pathology: a systematic review. Phys Ther. 2011;91(7):1079-1096.
  20. Nadler SF, Weingand K, Kruse RJ. The physiologic basis and clinical applications of cryotherapy and thermotherapy for the pain practitioner. Pain Physician. 2004;7(3):395-399.
  21. van der Windt DA, Simons E, Riphagen II, et al. Physical examination for lumbar radiculopathy due to disc herniation in patients with low-back pain. Cochrane Database Syst Rev. 2010;(2):CD007431.
  22. Walsh J, Flatley M, Johnston N, Bennett K. Slump test: sensory responses in asymptomatic subjects. J Man Manip Ther. 2007;15(4):231-238.
  23. Martin RL, Irrgang JJ, Sekiya JK. The diagnostic accuracy of a clinical examination in determining intra-articular hip pain for potential hip arthroscopy candidates. Arthroscopy. 2008;24(9):1013-1018.
  24. Clohisy JC, Knaus ER, Hunt DM, Lesher JM, Harris-Hayes M, Prather H. Clinical presentation of patients with symptomatic anterior hip impingement. Clin Orthop Relat Res. 2009;467(3):638-644.
  25. Modic MT, Masaryk TJ, Ross JS, Carter JR. Imaging of degenerative disk disease. Radiology. 1988;168(1):177-186.
  26. Chou R, Qaseem A, Owens DK, Shekelle P, Clinical Guidelines Committee of the American College of Physicians. Diagnostic imaging for low back pain: advice for high-value health care from the American College of Physicians. Ann Intern Med. 2011;154(3):181-189.
  27. Brinjikji W, Luetmer PH, Comstock B, et al. Systematic literature review of imaging features of spinal degeneration in asymptomatic populations. AJNR Am J Neuroradiol. 2015;36(4):811-816.
  28. Mullin BB, Mampilly G, Smith DC, Chamberlain BC, Heller JG. Imaging of the postoperative spine. Radiol Clin North Am. 2006;44(3):407-418.
  29. Qaseem A, Wilt TJ, McLean RM, Forciea MA, Clinical Guidelines Committee of the American College of Physicians. Noninvasive treatments for acute, subacute, and chronic low back pain: a clinical practice guideline from the American College of Physicians. Ann Intern Med. 2017;166(7):514-530.
  30. Ellis RF, Hing WA, McNair PJ. Comparison of longitudinal sciatic nerve movement with different mobilization exercises: an in vivo study utilizing ultrasound imaging. J Orthop Sports Phys Ther. 2012;42(8):667-675.
  31. Basson A, Olivier B, Ellis R, Coppieters M, Stewart A, Mudzi W. The effectiveness of neural mobilization for neuromusculoskeletal conditions: a systematic review and meta-analysis. J Orthop Sports Phys Ther. 2017;47(9):593-615.
  32. Clare HA, Adams R, Maher CG. A systematic review of efficacy of McKenzie therapy for spinal pain. Aust J Physiother. 2004;50(4):209-216.
  33. May S, Aina A. Centralization and directional preference: a systematic review. Man Ther. 2012;17(6):497-506.
  34. Bronfort G, Haas M, Evans R, Leininger B, Triano J. Effectiveness of manual therapies: the UK evidence report. Chiropr Osteopat. 2010;18:3.
  35. Hidalgo B, Detrembleur C, Hall T, Mahaudens P, Nielens H. The efficacy of manual therapy and exercise for different stages of non-specific low back pain: an update of systematic reviews. J Man Manip Ther. 2014;22(2):59-74.
  36. Rossi MD, Brown LE, Whitehurst M. Early strength response of the knee extensors during eight weeks of resistive training after unilateral total knee arthroplasty. J Strength Cond Res. 2005;19(4):944-949.
  37. Hodges PW, Richardson CA. Inefficient muscular stabilization of the lumbar spine associated with low back pain: a motor control evaluation of transversus abdominis. Spine. 1996;21(22):2640-2650.
  38. Johnson MI, Paley CA, Howe TE, Sluka KA. Transcutaneous electrical nerve stimulation for acute pain. Cochrane Database Syst Rev. 2015;(6):CD006142.
  39. Louw A, Zimney K, Puentedura EJ, Diener I. The efficacy of pain neuroscience education on musculoskeletal pain: a systematic review of the literature. Physiother Theory Pract. 2016;32(5):332-355.
  40. Sabnis AB, Diwan AD. The timing of surgery in lumbar disc prolapse: a systematic review. Indian J Orthop. 2014;48(2):127-135.
  41. Fritz JM, Childs JD, Wainner RS, Flynn TW. Primary care referral of patients with low back pain to physical therapy: impact on future health care utilization and costs. Spine. 2012;37(25):2114-2121.
  42. Dahlin LB, Lundborg G. The neurone and its response to peripheral nerve compression. J Hand Surg Br. 1990;15(1):5-10.
  43. Hall TM, Elvey RL. Nerve trunk pain: physical diagnosis and treatment. Man Ther. 1999;4(2):63-73.
  44. Konstantinou K, Dunn KM, Ogollah R, Vogel S, Hay EM, ATLAS study research team. Prognosis of sciatica and back-related leg pain in primary care: the ATLAS cohort. Spine J. 2018;18(6):1030-1040.
  45. Pincus T, Burton AK, Vogel S, Field AP. A systematic review of psychological factors as predictors of chronicity/disability in prospective cohorts of low back pain. Spine. 2002;27(5):E109-E120.
  46. Gellhorn AC, Katz JN, Suri P. Osteoarthritis of the spine: the facet joints. Nat Rev Rheumatol. 2013;9(4):216-224.
  47. Lewis R, Williams N, Matar HE, et al. The clinical effectiveness and cost-effectiveness of management strategies for sciatica: systematic review and economic model. Health Technol Assess. 2011;15(39):1-578.
  48. Jonsson B, Stromqvist B. Motor affliction of the L5 nerve root in lumbar nerve root compression syndromes. Spine. 1995;20(18):2012-2015.
  49. Williams MM, Hawley JA, McKenzie RA, van Wijmen PM. A comparison of the effects of two sitting postures on back and referred pain. Spine. 1991;16(10):1185-1191.
  50. Gordon SJ, Grimmer-Somers KA, Trott PH. Sleep position, age, gender, sleep quality and waking cervico-thoracic symptoms. Internet J Allied Health Sci Pract. 2007;5(1):1-8.
  51. O'Sullivan K, O'Keeffe M, Forster BB, Qamar SR, van der Westhuizen A, O'Sullivan PB. Managing low back pain in active adolescents. Best Pract Res Clin Rheumatol. 2019;33(1):102-121.
  52. White AA, Gordon SL. Synopsis: workshop on idiopathic low-back pain. Spine. 1982;7(2):141-149.
  53. Delitto A, George SZ, Van Dillen L, et al. Low back pain: clinical practice guidelines linked to the International Classification of Functioning, Disability, and Health from the Orthopaedic Section of the American Physical Therapy Association. J Orthop Sports Phys Ther. 2012;42(4):A1-A57.
  54. Mayer JM, Quillen WS, Verna JL, Chen R, Lunseth P, Dagenais S. Impact of a supervised worksite exercise program on back and core muscular endurance in firefighters. Am J Health Promot. 2015;29(3):165-172.
  55. Germon T, Ahuja S, Casey ATH, Todd NV, Rai A. British Association of Spine Surgeons standards of care for cauda equina syndrome. Spine J. 2015;15(3):S2-S4.
  56. Ahn UM, Ahn NU, Buchowski JM, Garrett ES, Sieber AN, Kostuik JP. Cauda equina syndrome secondary to lumbar disc herniation: a meta-analysis of surgical outcomes. Spine. 2000;25(12):1515-1522.
  57. Gibson JN, Waddell G. Surgical interventions for lumbar disc prolapse: updated Cochrane Review. Spine. 2007;32(16):1735-1747.
  58. North American Spine Society. Evidence-based clinical guidelines for multidisciplinary spine care: diagnosis and treatment of low back pain. Burr Ridge (IL): North American Spine Society; 2020.
  59. Hoffman RM, Wheeler KJ, Deyo RA. Surgery for herniated lumbar discs: a literature synthesis. J Gen Intern Med. 1993;8(9):487-496.
  60. Rasouli MR, Rahimi-Movaghar V, Shokraneh F, Moradi-Lakeh M, Chou R. Minimally invasive discectomy versus microdiscectomy/open discectomy for symptomatic lumbar disc herniation. Cochrane Database Syst Rev. 2014;(9):CD010328.
  61. Atlas SJ, Keller RB, Wu YA, Deyo RA, Singer DE. Long-term outcomes of surgical and nonsurgical management of lumbar spinal stenosis: 8 to 10 year results from the Maine lumbar spine study. Spine. 2005;30(8):936-943.
  62. Weinstein JN, Tosteson TD, Lurie JD, et al. Surgical vs nonoperative treatment for lumbar disk herniation: the Spine Patient Outcomes Research Trial (SPORT): a randomized trial. JAMA. 2006;296(20):2441-2450.
  63. McGregor AH, Dore CJ, Morris TP. Function after spinal treatment, exercise and rehabilitation (FASTER): improving the functional outcome of spinal surgery. BMC Musculoskelet Disord. 2010;11:17.
  64. Choi BK, Verbeek JH, Tam WW, Jiang JY. Exercises for prevention of recurrences of low-back pain. Cochrane Database Syst Rev. 2010;(1):CD006555.
  65. Yeoman W. The relation of arthritis of the sacro-iliac joint to sciatica, with an analysis of 100 cases. Lancet. 1928;212(5492):1119-1122.
  66. Marras WS, Lavender SA, Leurgans SE, et al. The role of dynamic three-dimensional trunk motion in occupationally-related low back disorders: the effects of workplace factors, trunk position, and trunk motion characteristics on risk of injury. Spine. 1993;18(5):617-628.
  67. Shiri R, Karppinen J, Leino-Arjas P, Solovieva S, Viikari-Juntura E. The association between obesity and low back pain: a meta-analysis. Am J Epidemiol. 2010;171(1):135-154.
  68. Mok NW, Brauer SG, Hodges PW. Hip strategy for balance control in quiet standing is reduced in people with low back pain. Spine. 2004;29(6):E107-E112.
  69. Huang W, Qian Y, Zheng K, Yu L, Yu X. Is smoking a risk factor for lumbar disc herniation? Eur Spine J. 2016;25(1):168-176.
  70. Gordon R, Bloxham S. A systematic review of the effects of exercise and physical activity on non-specific chronic low back pain. Healthcare (Basel). 2016;4(2):22.
  71. Cramer H, Lauche R, Haller H, Dobos G. A systematic review and meta-analysis of yoga for low back pain. Clin J Pain. 2013;29(5):450-460.

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